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Robert Raike Ph.D. 2004 Thesis Title: A Potential Mouse Model of Episodic Ataxia Type 2 Advisor: Christopher M. Gomez

Thesis Publications:

Raike RS, Kordasiewicz HB, Thompson RM, Gomez CM. Dominant-negative suppression of Ca(v)2.1 currents by alpha(1)2.1 truncations requires the conserved interaction domain for beta subunits. Molecular Cellular Neuroscience. 2007 Feb;34(2):168-77.

Subramony SH, Schott K, Raike R, Callahan J, Langford L, Christova P, Anderson J H, Gomez C M (2003). Novel CACNA1A mutation causes febrile episodic ataxia with interictal ocular motor and posture abnormalities. Annals of Neurology. 4(6): 725-31

Restituito S, Thompson RM, Eliet J, Raike RS, Riedl M, Charnet P, Gomez CM. (2000) The polyglutamine expansion in spinocerebellar ataxia type 6 causes a beta subunit-specific enhanced activation of P/Q-type calcium channels in Xenopus oocytes. Journal of Neuroscience. 20(17): 6394-403.

Other Publications:

Raike RS, Jinnah HA, Hess EJ (2005). Animal Models of Generalized Dystonia. NeuroRx. 2(3): 504-12

Weisz CJC, Raike RS, Soria-Jasso LE, Hess EJ (2005). Potassium channel blockers inhibit the triggers of attacks in the calcium channel mouse mutant tottering. Journal of Neuroscience. 24(16): 4141-45.